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Wayne Shared "Pathology Bliss 6/15/2024" - 53 Picmonics

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Pathology Bliss 6/15/2024

Henoch-Schonlein Purpura
Most Common Childhood Vasculitis
Follows Pharyngeal or Respiratory Infection
Palpable Purpura on Legs and Buttocks
Arthralgia
Intestinal Hemorrhage
Black Stool (Melena)
Associated with IgA Nephropathy
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2 mins
Berger's Disease (IgA Nephropathy)
Nephritic
IgA Nephropathy
Immune Complex Deposit in Mesangium
Increased Synthesis of IgA
Most Common Cause of Nephritic Syndrome
Overlapping Features with Henoch–Schönlein Purpura
Presents with Pharyngitis, URI or Acute Gastroenteritis
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1 min
Membranoproliferative Glomerulonephritis
Nephritic-Nephrotic Syndrome
Type I
Subendothelial Immune Complexes
Hepatitis C Virus
Granular IF
Tram Tracks Caused by Splitting of the GBM
Ingrowth of Mesangium
Type II
Dense Deposit Disease
C3 Nephritic Factor
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2 mins
Celiac Disease Characteristics
Epidemiology
European Descent
HLA-DQ2
HLA-DQ8
Pathophysiology
Autoimmune-mediated Intolerance of Gliadin
Symptoms
Dermatitis Herpetiformis
Weight Loss
Failure to Thrive
Decreased Bone Density
Steatorrhea
Anemia
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2 mins
Post Streptococcal Glomerulonephritis
Pathophysiology
Nephritic
Children
Signs and Symptoms
Edema
Cola Colored Urine
Follows Group A Streptococcal pharyngitis or Skin Infection
Diagnosis
Neutrophils
Immunofluorescence (IF) Granular
Lumpy Bumpy Appearance on Immunofluorescence (IF)
Subepithelial Immune Complex Humps
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2 mins
Rapidly Progressive Glomerulonephritis
Pathogenesis
Nephritic Syndrome
Crescent Shape
Crescents Consist of Fibrin and C3b
With Parietal Cells, Monocytes, and Macrophages
IMMUNOFLUORESCENCE
Linear Immunofluorescence
Negative Immunofluorescence
Granular Immunofluorescence
Etiology
Goodpasture Syndrome
Granulomatosis with Polyangiitis (Formerly Wegener's)
Microscopic Polyangiitis
Prognosis
Poor Prognosis
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3 mins
Alport Syndrome
Pathophysiology
Nephritic
X-linked
Mutation in Type IV Collagen
Diagnosis
Split Basement Membrane
Basket Weave Appearance
Signs and Symptoms
Ocular Disorders
Deafness
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2 mins
Membranous Glomerulonephritis
Characteristics
Nephrotic
Diagnosis
LM Thickening of Capillary and Glomerular Basement Membrane (GBM)
EM Spike and Dome Appearance with Subepithelial Deposits
IF Granular
Primary
Phospholipase A2 Receptor Autoantibodies
Secondary
NSAIDS
Gold
Solid Tumors
Hepatitis B Virus (HBV)
Lupus
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3 mins
Minimal Change Disease
Pathophysiology
Nephrotic
Most Common in Children
May be Triggered by Recent Infection or Immune Stimulus
Diagnosis
On Electron Microscopy (EM) See Foot Process (Podocyte) Effacement
On Light Microscopy (LM) See Normal Glomeruli
Loss of Negative Charge
Selective Loss of Albumin
Treatment
Responds to Corticosteroids
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2 mins
Diabetic Glomerulonephropathy
Pathophysiology
Nonenzymatic Glycosylation of Glomerular Basement Membrane
Nonenzymatic Glycosylation of Arterioles
Hyperfiltration
Increased GFR
Histological Findings
Hyaline Arteriolosclerosis
Mesangial Expansion
Type IV Collagen Deposition
Kimmelstiel Wilson Lesions
Presentation
Nephrotic
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2 mins
Diffuse Proliferative Glomerulonephritis (DPGN)
Nephritic and Nephrotic Syndrome
Associations
Systemic Lupus Erythematosus (SLE)
IgA Nephropathy
Characteristics
Wire Looping
IF Granular
Subendothelial Immune Complexes
Subepithelial Immune Complexes
C3 Deposition
Treatment
Treat Underlying Disorder
Steroids
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2 mins
Prerenal Acute Kidney Injury Pathophysiology and Presentation
Pathophysiology
Decreased Renal Perfusion
Decreased GFR
Increased Sodium and Water Reabsorption
Presentation
Edema
Dyspnea
Hypotension with Tachycardia
Altered Mental Status
Oliguria
Etiologies
NSAIDs
Renal Artery Stenosis
ACE Inhibitors
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2 mins
Chronic Kidney Disease Early Symptoms Assessment
Characteristics
GFR < 60mL/min
Accumulation of Waste Products
Clinical Features
General Malaise
Hypertension
Proteinuria
Hyperkalemia
Mineral and Bone Disorders
Neuropathy
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2 mins
Acute Tubular Necrosis
ETIOLOGY
Ischemic Injury
Nephrotoxic Injury
HISTOLOGY
Granular Muddy Brown Casts
Signs & Symptoms
Intrinsic Renal Failure
Oliguric Phase
Metabolic Acidosis
Hyperkalemia
Increase in BUN and Creatinine
Polyuria Phase
Decrease in BUN and Creatinine
Hypokalemia
Treatment
Supportive Care
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3 mins
Chronic Kidney Disease Late Symptoms Assessment
Metabolic Acidosis
Severe Uremia
Arrhythmias
Edema
CNS Depression
Anemia
Oliguria
Pruritus
Considerations
End Stage Renal Disease (ESRD)
GFR < 15mL/min
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2 mins
Chronic Kidney Disease Interventions
Interventions
Daily Weights
Strict I/O
Renal Diet
Strict Medication Regimen
Erythropoietin
Manage Hyperkalemia
Manage CKD-MBD
Dialysis
Kidney Transplant
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2 mins
Renal Osteodystrophy
Pathophysiology
Chronic Kidney Disease
Secondary Hyperparathyroidism
Hyperphosphatemia
Hypocalcemia
Decreased Calcitriol
Clinical Features
Osteitis Fibrosa Cystica
Osteoporosis
Osteomalacia
Management
Treating Underlying Disorder
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2 mins
Prerenal Acute Kidney Injury Diagnosis and Management
Diagnostic Findings
BUN:Creatinine Ratio > 20:1
Fractional Excretion of Sodium (FENa) < 1%
Urine Osmolality > 500 mOsm/kg
Hyaline Casts
Management
Fluid Replacement
Monitor Renal Function
Diuretics
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2 mins
Postrenal Acute Kidney Injury Pathophysiology and Presentation
Pathophysiology
Bilateral Outflow Obstruction
Decreased GFR
Etiologies
Benign Prostatic Hyperplasia (BPH)
Nephrolithiasis
Neoplasm
Neurogenic Bladder
Presentation
Bladder Distension
Lower Abdominal Pain
Edema
Oliguria
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3 mins
Post Renal Acute Kidney Injury (AKI) Diagnosis and Management
Diagnosis
Physical Examination
BUN:Creatinine ratio of 20:1 in the Initial Stages
BUN:Creatinine ratio of 10:1 in the late stages
Urine Osmolality <350 mOsm/kg
Variable Fractional Excretion of Sodium (FENa)
Ultrasound
CT Scan
Management
Treat Underlying Cause
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4 mins

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